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On a molecular level, this may also occur through mitochondrial dysfunction associated with decreased fat degradation due to PTM-mediated suppression of the enzymes in -oxidation [103, 211], upregulated immune cell infiltrations, protein adducts, lipid peroxidatifon, and DNA damage [190].Alcohol-mediated ROS and RNS can also downregulate -oxidation activity by inhibiting peroxisome proliferator-activated receptor- (PPAR-) and a lipid catabolism regulator named AMP-activated protein kinase (AMPK) [9, 213] while upregulating sterol regulatory binding protein-1 (SREBP-1) to increase hepatic fatty acid and cholesterol biosynthesis [9, 213]

439 These data suggest that lipid peroxidation is a major factor in the progression of Friedreichs Ataxia

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Bacterial outer membrane vesicles mediate cytosolic localization of LPS and caspase-11 activation

10.1186/1479-5876-9-137 18 KlasseP