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However, on the TNF flow cell surface, all dosed subjects in cohort 7 showed increasing signal compared to baseline which was confirmed with the anti-human IgG reagent, but not the AMG 966-specific antibody 1A3, indicating these subjects developed endogenous anti-TNF antibodies as a result of AMG 966 treatment ( Figures 8F, H )

Epithelial production of elastase is increased in inflammatory bowel disease and causes mucosal inflammation

Synthesis of 3-keto-4-diazo-5--dihydrosteroids as potential irreversible inhibitors of steroid 5--reductase.Tetra.Lett.1980,21, 15-18 75

In part two, again serum antibodies bound to the immobilized protein and signal enhancement with mAb 1A3 (specific to AMG 966) was used in place of anti-human IgG

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