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Hepatotoxicity of ketoconazole in Sprague-Dawley rats: glutathione depletion, flavin-containing monooxygenases-mediated bioactivation and hepatic covalent binding

Most patients feel comfortable returning to work and social activities

112 Unfortunately, in this and a few other case reports of severe secondary oxalosis, no details were provided regarding the potential presence of a loss-of-function mutation in genes involved in primary hyperoxaluria in these patients

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In fact, each phenomenon is necessary for the induction or amplification of the other