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Haploinsufficiency of the SLC52A1 (OMIM#615026) due to maternal microdeletion and heterozygous intronic variant has been reported to cause a transient riboflavin responsive neonatal multiple acyl-CoA dehydrogenase deficiency that resolved with oral supplementation of riboflavin [27-29]

To deplete their increased concentration, there are so many endogenous as well as exogenous antioxidants are available, including both enzymatic and non-enzymatic antioxidants