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Delivery-mediated exosomal therapeutics in ischemia-reperfusion injury: advances, mechanisms, and future directions

To evaluate whether mutants deficient in different glutathione-dependent enzymes are impacted by TMHT, we subjected cytosolic glutaredoxin mutants to TMHT (Figure 4C)

They uncovered a new type of cell death, whereby mitochondria move to the plasma membrane generating ROS, which in turn damages the plasma membrane leading to cell lysis, the type of lysis being termed mitoxyperilysis (Fig

Cancer mortality and senescence: Is redox therapy an option

4-hydroxynonenal induces apoptosis via caspase-3 activation and cytochrome c release