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Prevalence of atopic comorbidities in eosinophilic esophagitis: A case-control study of 449 patients

Notably, trophoblast cells may respond to maternal immune dysregulation by upregulating Fas ligand (FASL) and tumor necrosis factor receptors (TNFRs), thereby triggering apoptotic signaling and promoting trophoblast dysfunction (51)

(2012), is characterized by iron-dependent accumulation of lipid peroxides, distinguishing it from classical cell death modalities such as apoptosis, necrosis, and autophagy, ferroptosis exhibits a stronger dependence on intracellular iron than other regulated cell death pathways

We assume that at least two Cys residues of GSSAT are oxidized, which may form an intracellular disulfide bond under oxidizing conditions

doi: 10.1007/978-3-030-51652-9_16 39 LuoYWangHLiuBWeiJ