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Because NNMT consumes nicotinamide, its inhibition theoretically increases the availability of nicotinamide for entry into the NAD + salvage pathway via nicotinamide phosphoribosyltransferase (NAMPT)

Additionally, vascular resistance is decreased following the direct deactivation of the beta-adrenoceptors on the juxtaglomerular apparatus [34]

in the absence of other abnormal test results) reduces within 1224 hours of intravenous administration of vitamin K, then the prolonged PT can be explained by vitamin K deficiency[15]

International Journal of Cardiology 107.1 (2006): 54-60