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To investigate whether VRK2 directly phosphorylates Sec24C, we carried out an in vitro kinase assay and found that overexpression of VRK2 was able to increase the phosphorylation level of Sec24C

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These components, such as mtDNA, mitochondrial RNA (mtRNA), mitochondrial ROS (mtROS), and N-formyl peptides, can be released upon mitochondrial stress or damage