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Secondary carnitine deficiency can be caused by increased losses, pharmacological therapy, a number of inherited metabolic disorders [38], poor diet or malabsorption of carnitine, from increased renal tubular loss of free carnitine (Fanconi syndrome), haemodialysis, peritoneal dialysis, or the increased excretion of acylcarnitines[39] with certain drugs

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2, COE treatment extended the anagen phase in a dose-dependent manner

Clin Ter

Medications like ranitidine and finasteride, as FMO substrates, competitively inhibit TMA binding, consequently diminishing TMAO generation and potentially mitigating CKD progression