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This promoted Peroxisome proliferator activated receptor (PPAR) ubiquitination and degradation, leading to the inhibition of fatty acid metabolism

2004;286:R505R511

327 316 10.1016/S0076-6879(00)27263-7 [DOI] [PubMed] [Google Scholar] Gaberc-Porekar V., Menart V

( 2 ) The observed cytoplasmic translocation of p53 during co-treatment with 4-OH-E 1 is due to the maintenance of the SIRT1 protein level and ultimately its function in glutamate-treated neuronal cells

33 Collectively, these existing studies were generally consistent and revealed the course of blood-toward-BALF transition and the contribution of pulmonary monocytemacrophages to CS via the release of multiple pro-inflammatory cytokines and chemokines during severe COVID-19