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Because this high antioxidant capacity enhances cell survival and impairs cellular responses to anticancer therapy (79), induction of ROS-mediated damage in cancer cells with use of appropriate pharmacological agents that either promote ROS generation beyond the cellular anti-oxidative capacity or disable the cellular antioxidant system, has been considered as a radical therapeutic strategy for preferential targeting of cancer cells (79)

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CrossRef Efficacy and safety of glucagon-like peptide-1 (GLP-1) agonists in pediatric obesity: A systematic review and meta-analysis Ghassan A

Missing appeal deadlines forfeits the right to challenge denials and shifts financial responsibility to the practice or patient