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The ternary complex of an inactive mutant of the enzyme with bound NADPH and 5-fluorouracil reveals the architecture of the substrate-binding sites and residues responsible for recognition and binding of the drug

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As epitalon has been shown to bind to H1 (Khavinson et al 2020) it is possible that peptide may downregulate telomerase in cancer cells (through H1 binding and H19 upregulation), and activate ALT (through protein trapping) at the same time

Group II was induced with TBI but not treated while Group III was neither induced with TBI nor treated

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