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Mechanistic pathways associated with acetaminophen-induced hepatotoxicity APAP is rapidly absorbed and extensively metabolized upon entering the liver, with 8590% conjugated with glucuronic acid or sulfate by the enzymes uridine diphosphate-glucuronosyltransferase and sulfotransferase and subsequently eliminated

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However, there is no scientific basis for this

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Clinical studies of ASD and experiments on animal and cellular models of this disorder have revealed the biochemical endophenotype of insufficient mitochondrial energy production