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Thus, POAG progression seems to be associated with the accumulation of zinc-amino acid complexes, especially in the early stages of the disease

Although in both disorders the immune response is different, genetically susceptible individuals have an inappropriate mucosal immune response against their gut microbiota, mediated by an excessive activation of effector T cell subsets and/or a deficiency of Treg cells (65) (Figure 1A)

Juvenile parkinsonism associated with heterozygous frameshift ATP13A2 gene mutation

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It also reduced pro-inflammatory cytokines (IL-6, TNF-, IFN-) and the expression of the p38 MAPK pathway