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As a critical signaling molecule, IGF-I triggers the proliferation and differentiation of cardiac fibroblasts by activating various signaling pathways

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Like hexokinase, PFK-1 requires ATP as a substrate and catalyzes an essentially irreversible reaction

The development of the hepatic steatosis is due to the imbalance between the capacities of synthesis and export of lipids by hepatocytes

This regenerative effect becomes more pronounced with longer treatment periods, suggesting that TB-500 works through fundamental tissue repair rather than temporary symptomatic improvement