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The exceptional in vitro potency is attributed to its mechanism of acting as an allosteric potentiator of HGF binding to c-Met (amplifying an endogenous signal), which produces non-linear dose-response curves

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Finally, excessive glucose flux through the hexosamine pathway leads to UDP-GlcNAc accumulation and subsequent O-GlcNAcylation of serine/threonine residues on antioxidant enzymes, like SOD, reducing their activity (Paneque et al., 2023)

aeruginosa clearance by promoting NOX2-dependent ROS production To investigate the contribution of NADPH oxidase-derived ROS to MPLA-induced antimicrobial immunity, we first assessed activation of the NADPH oxidase complex, a major source of ROS in macrophages, after MPLA treatment