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In the gut, T cells maintain immune balance, differentiating into proinflammatory (Th1, Th17) or anti-inflammatory [Th2, regulatory T cells (Tregs)] subsets (Dressman and Elyaman, 2022)

The other argument is that gout is primarily an inflammatory disease resulting in systemic increases in proinflammatory factors 20

Consequently, mitochondrial metabolism and signaling including the UPR mt have been implicated in integrating nutrient- and microbiota-derived signals in the intestinal stem cell niche 8 , critically affecting the regenerative capacity of the epithelium under homeostatic and disease conditions

2017 Nov 22;359:j5024