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Methods: We reprogrammed iPSCs from a HT1 patient

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Despite this, both SPR and western blot analysis data revealed significant reductions in A binding, strongly suggesting that this observed decrease in A binding did not originate from changes in the sialylation status of ApoE

Mitochondrial transfer has been demonstrated to restore oxidative phosphorylation, reprogram redox balance, and support tumor proliferation and metastasis in the tumor context [19]

Patients with fatty liver disease were classified to non-advanced fibrosis (fibrosis stage 0-2) and advanced fibrosis (stage 3-4)