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CAF-derived exosomes have been shown to contain amino acids, lipids and TCA intermediates that inhibit mitochondrial oxidative phosphorylation, thereby increasing glutamine-dependent reductive carboxylation and promote glycolysis in tumor cells [149]

We next wanted to confirm that these mTORC1- and ATF4-induced changes were independent of the ISR

The term mTORopathy is used in the context of neurological diseases, especially epileptic encephalopathies and cortical malformations such as tuberous sclerosis due to mutations in TSC1 or TSC2

Four prominent mechanisms by which mitochondria communicate with the rest of the cell include the release of cytochrome c to induce cell death, activation of AMP-activated protein kinase (AMPK) to control mitochondrial fission and fusion, production of reactive oxygen species (ROS) to activate transcription factors, and the release of mitochondrial DNA (mtDNA) to activate immune responses 2,3,4,5 (Fig

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