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Because previous studies showed marked differences between MET phosphorylation levels and the strength of MET-specific phenotypes induced by HGF or HGF mimics 49 , we further analyzed the capacity of the different K1 proteins to trigger the scattering of human cells in vitro (Fig

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TDP-43 repression of nonconserved cryptic exons is compromised in ALS-FTD

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L.OppoV.FabbriM.SarchiotoM.et al

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Mechanism of ubiquitin activation revealed by the structure of a bacterial MoeBMoaD complex

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