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This susceptibility allows ferroptosis to be targeted as a potential vulnerability in therapeutic strategies [18, 19]

Zinc transport from the endoplasmic reticulum to the cytoplasm via Zip7 is necessary for barrier dysfunction mediated by inflammatory signaling in RPE cells

In Managing As in the Environment: From Soil to Human Health, Edited by: Naidu, R., Smith, E., Owens, G., Bhattacharya, P

As a substrate for glutathione S-transferase, this agent reacts with a number of harmful chemical species, such as halides, epoxides and free radicals, to form harmless inactive products

Unlike conventional T cells, T cells recognize and eradicate transformed cells in a major histocompatibility complex (MHC)-independent manner, positioning them as ideal candidates to target MHC-deficient CSCs 10,11,12