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Simultaneously, depletion of intracellular GSH, dysregulation of Nrf2 signaling, and iron-dependent lipid peroxidation weaken endogenous defenses, promoting neuronal death and contributing to ferroptosis pathways

This indicates that the oxidation of PUFA by LOXs through a PHKG2-dependent iron pool is required for ferroptosis, and that the covalent inhibition of the catalytic selenocysteine in GPX4 prevents the elimination of PUFA hydroperoxides

Nat Rev Immunol (2010) 10(3):2105

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By bolstering your bodys natural detoxification processes, glutathione contributes to a healthier liver and overall well-being