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KEAP1/NRF2 mutant cells are protected against cysteine-starvation induced ferroptosis due to elevated GCLC expression and limited glutamate accumulation, and dual targeting of cystine import and GCLC activity can overcome the resistance of NRF2 active cells to ferroptosis (Kang et al., Cell Metab 2021)

PMID 15474517

The consumption of seaweed as a protective factor in the etiology of breast cancer: Proof of principle

2005 Jan-Feb;7(1):27-31

In summary, these results further support the hypothesis that IFN signaling can promote RMC tumor cell state changes, prompting further investigation into the pharmacological inhibition of myeloid-affiliated mediator, p300, which is downstream of IFNGR1