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To avoid massive overproduction of proteins in women that could potentially be lethal, the female body has a unique mechanism for shutting down protein production in one of the XX pair

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These class II CFTR variants are associated with altered structural conformation, and this misfolded CFTR proteins is degraded by the chaperons mediated Endoplasmic Reticulum-Associated Degradation (ERAD) pathway

At the same time, the article analyzes the profound influence of various factors in the TME (such as metabolic stress, immune suppression networks, and stromal interaction interfaces) on the process of T cell exhaustion