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2022 Apr;40:99-109

2.1.2 Vesicular monoamine transporter 2 inhibition METH inhibits vesicular monoamine transporter 2 and disrupts monoamine storage in synaptic vesicles (Lizarraga et al., 2015)

However, there is little evidence for greater benefit of slow-release preparations

2.1.4 Improvement of homocysteine-induced endothelial dysfunction Hyperhomocysteinemia (HHcy) is recognized as an independent risk factor for cardiovascular disease