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Thus, this study revises the conventional doctrine of GSH function in protein redox regulation and liver pathophysiology, indicating that hepatic GSH depletion by Chac1 overexpression may represent a potentially novel therapeutic approach for MASLD (Figure 6G)

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Sun TC, Liu XC, Yang SH et al (2020) Melatonin inhibits oxidative stress and apoptosis in cryopreserved ovarian tissues via Nrf2/HO-1 signaling pathway

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