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10.4161/auto.5173 Autophagy 24 ConnorJ

This product is intended exclusively for research, analytical, and educational applications

Neuroscience 145 , 185196 (2007)

GO enrichment analysis showed that the upregulated DEGs were mainly enriched in apoptosis, inflammation, and oxidative stress, while the downregulated DEGs were primarily involved in gap junctions, cell proliferation, and sex hormone response (Figure 3B), suggesting that the damage to granulosa cell function, including increased apoptosis and decreased proliferation ability, may be a critical cause of follicular atresia and oocyte maturation disorders

The negative effects of Ang II/AT1 pathway in Ecs can be counteracted by the Ang (1-7)/Mas pathway