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BTW - This is a lot of info

Plant and animal toxins

It has been observed that the conversion from 2-hydroxysetrogens to 4-hydroxyestrogen might be an additional pro-inflammatory signal because these endogenous metabolites can be oxidized to 3,4-quinone, which lead to depurination and mutation [49]

Researchers originally investigated this fragment as part of efforts to isolate the lipolytic activity of hGH without the proliferative or metabolic side effects associated with the full molecule

Heres the whole thing in plain terms: What they did: built and validated a lab-grade method to accurately measure 5A1MQ in the blood, then dosed rats two ways intravenous (IV) and oral and tracked exactly how much showed up in circulation over time