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Impact of N-acetylcysteine on endothelial function, B-type natriuretic peptide and renal function in patients with the cardiorenal syndrome: a pilot cross over randomised controlled trial

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Thymic atrophy, driven by immunosenescence as well as endogenous factors (such as elevated levels of glucocorticoids and sex steroids) and exogenous insults (including chemotherapy, irradiation, malnutrition, chronic inflammation, and infections), ultimately reduces the thymic output of newly generated T cells [82, 83]

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