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The cell cycle, as well as senescence, can be regulated by metabolism and mitochondrial activity through p53

In ovarian cancer (IOSE144, A2780, OVCAR3, and SK-OV-3) cells, dihydroartemisinin downregulates Bcl-2 and upregulates Bax, which inhibits cancer cell proliferation and leads to cell cycle arrest at the S phase [104, 147, 148]