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Egr, Nos, Srf, Vegfr, Akt1, Plc, and Kras ) provide a likely special point to explain how the dysfunction and its counteraction is causal to, or result of (Vukojevic et al., 2018) (see Genes Expression as Support )

Dihexa-mediated protection is attenuated by co-treatment with the HGF antagonist 6-AH, further evidence that HGF activation is a component of the observed protection