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Indeed, accumulating evidence based on preclinical studies has identified ERK5 activation as a mediator of resistance to vemurafenib (BRAFi) and trametinib (MEKi) [54], and therefore, the combined targeting of ERK5 and ERK1/2 pathways results in better outcomes [55]

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Anoxic depolarization, which occurs during ischemia, triggers ATP release from neurons and glial cells leading to a marked elevation of extracellular ATP concentration (Dale and Frenguelli, 2+ overload, tissue excitotoxicity, and amplification of the inflammation (Domercq et al., 2018)

This disruption allows blood-derived proteins, typically excluded from the brain parenchyma, to infiltrate the central nervous system (CNS) [53,54,55,56]

Applying machine learning to identify autistic adults using imitation: an exploratory study