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On the other hand, p53 inhibits ferroptosis by inhibiting the activity of dipeptidyl peptidase 4 (DPP4) or inducing the expression of cyclin-dependent kinase inhibitor 1A (CDKN1A/p21) [77], both indicate the importance of p53 as a regulator of metabolism-related genes in ferroptosis (Fig

PHCs provided care to HCV treatment-nave patients with no or mild fibrosis (FIB-4 score 2-fold increased DAA susceptibility were analyzed

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In our study, metabolomics based on UPLC-Orbitrap-MS/MS analysis method was applied to investigate the changes in fecal metabolites

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