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Thus, TNBS promotes two independent mechanisms of injury, GSH depletion and ROS generation, both being required for the manifestation of mucosal injury as GSH limitation renders intestine susceptible to the TNBS-induced ROS overgeneration

aureus is sufficient to initiate autophagy, which can support the bacterial replication with host cells

Every department has its own practices in place

Thiamine and quinine differently inhibit the early phase of acetylcholine-dependent contraction of mouse ileum in vitro

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