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Sex differences in non-alcoholic fatty liver disease: hints for future management of the disease

This leaves our cells more vulnerable to damage

Beyond its well-established proinflammatory actions, HMGB1 functions as a redox-sensitive protein that dimerizes under oxidative stress to protect adjacent DNA from hydroxyl radical-induced damage[60]

DFO chelates ferric iron to limit iron-mediated ROS production by the Fenton reaction, which promotes lipid peroxidation in ferroptosis [28]

Interestingly, an oxyntomodulin multi-agonist was under investigation concurrently with the GLP1/glucagon multi-agonist

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