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(2015) reported that both TRPV1 knockout and pharmacological inhibition of the channel in an ex vivo model of increased IOP, resulted in fewer RGCs labeled for an apoptotic marker, suggesting that the entry pathway of TRPV1 Ca 2+ could be related to cell death (Sappington et al., 2015)

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4) Results were then integrated and compared to literature findings

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In addition to their role in modulating signal transduction, fatty acids serve as a vital source of cellular energy that sustain cell growth, survival, and function through fatty acid -oxidation

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Confirming Gilbert syndrome involves genetic testing to detect a specific gene mutation

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The siderophore transporter Sit1 determines susceptibility to the antifungal VL-2397

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