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This paper demonstrates the importance of dysregulation of stress granule dynamics and its interaction with nucleocytoplasmic transport as a pathophysiological mechanism contributing to motor neuron injury in models of ALS

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On the other hand, under the regulation of various cells and their secreted cytokines, key transcriptional regulation pathways such as TLR2/4-NF-B, p38/MAPK, IL-6/STAT3, and others participate in the immune-inflammatory interactions underlying diabetic vascular complications, ultimately leading to vascular damage and barrier disruption, triggering diabetes-related macrovascular and microvascular complications (Odegaard et al., 2016