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Description

vulgaris suppresses the phosphorylation of p65 which act as a resistant mechanism to TRAIL [64]

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IFN-, IL-17A, or zonulin rapidly increase the permeability of the blood-brain and small intestinal epithelial barriers: relevance for neuro-inflammatory diseases

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A considerable percentage of the worlds population has at least one variant of this gene

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We therefore employed extrachromosomal DSB substrates that could be electroporated into mouse fibroblasts (Fig

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doi: 10.1002/humu.21399

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