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doi: 10.1016/j.neuroscience.2013.03.002 Widdowson PS, Farnworth MJ, Upton R, Simpson MG, 1996

Modulating macrophage polarization, inhibiting pro-inflammatory signaling pathways, and promoting tissue repair mechanisms are potential approaches for attenuating inflammation, reducing tissue damage, and improving lung function in individuals with COPD

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In this context, it should also be mentioned that bacterial metabolites from dAGEs-induced gut dysbiosis may also contributed to augmented inflammation and Th2 immunity by activating the TLR4 pathway post-circulatory entry