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Sinomenine can phosphorylate p62 Ser351 to degrade Keap1 and increase Nrf2 expression, and play a role in protecting bone destruction by increasing p62 expression and activating the p62-Keap1-Nrf2 axis through p62 Thr269/Ser272 phosphorylation

In addition, when all patients with MDD were divided into the NDT group, which is a group of patients without drug treatment, and the DT group, which received drug treatment, and the difference from the control group was compared (Figures 5B,C), acetylcarnitine was decreased in the MDD patient group compared to that in the control group

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This metabolic shift has been well documented in previous studies, with findings indicating that LC supplementation can improve the rate of fatty acid oxidation and reduce the use of muscle glycogen, ultimately improving performance during prolonged exercise bouts