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These findings highlight the critical role of THC-induced oxidative stress as a contributing factor to cannabinoid-mediated neuropsychiatric risk and identifies a novel antioxidant treatment candidate for the prevention and/or reversal of these pathophysiological outcomes

Statistical analysis Statistical analysis was performed via two-tailed Students t -tests, one-way ANOVA and two-way ANOVA via GraphPad Prism

To evaluate protection against acute injury without persistent triggers, we employed a radiation enteritis (RE) model with single-dose irradiation (Supplementary Fig

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