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Furthermore, cardamomin suppressed the phosphorylation of Akt, a protein that regulates the NF-B transcription through a mechanism dependent on RelA/p65 phosphorylation

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Furthermore, excessive ROS generation can disrupt nitric oxide (NO) homeostasis by reducing endothelial nitric oxide synthase (eNOS) activity, while upregulating inducible nitric oxide synthase (iNOS), leading to impaired vasodilation and exacerbated venous stasis [11]

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