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Human amylin has a strong tendency to aggregate into amyloid fibrils , similarly to amyloid in Alzheimers or -synuclein in Parkinsons

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Current research indicates that the cell cycle arrest depends on the activity of cyclin-dependent kinase inhibitors (CDK) p16 and p21, often regulated by the main tumor suppressor protein p53, whose elevated level is frequently used as a marker of senescence detection [21]

DOI: 10.3390/biotech14020041, Costa, J