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Molecular heterogeneity and receptor coamplification drive resistance to targeted therapy in MET-amplified esophagogastric cancer

Repeated freeze-thaw cycles should be strictly avoided, as these degrade peptide integrity and introduce unwanted variability into experimental results

Preclinical studies have explored its involvement in repair processes, vascular adaptation, and inflammatory regulation, positioning it as a molecule of interest in musculoskeletal and gastrointestinal research contexts (1)