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doi: 10.1111/all.v74.12 106 AkdisMAabAAltunbulakliCAzkurKCostaRACrameriRet al

To predict further players in septal fibrosis formation, DT-simulations upon perturbations (e.g

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In cultured hepatic HepG2 cells, it ameliorated NASH by inhibiting nucleotide-binding oligomeric structural domains, NODlike receptors family pyrin domain containing 3 (NLRP3), and focalization via mitotic activation [171]

It also forms actin tails, causing cell-to-cell spread, thus allowing the bacterium to survive the extracellular environment and evade both host defenses and antibiotic treatment [58]