oxygen dependent intracellular killing ros production glutathione Toxicity and Reactive Oxygen Species: The Devil Is in the Details Neutrophils to the ROScue: Mechanisms – Mitochondrial Glutathione in Cellular Redox

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Because this high antioxidant capacity enhances cell survival and impairs cellular responses to anticancer therapy (79), induction of ROS-mediated damage in cancer cells with use of appropriate pharmacological agents that either promote ROS generation beyond the cellular anti-oxidative capacity or disable the cellular antioxidant system, has been considered as a radical therapeutic strategy for preferential targeting of cancer cells (79)

Animals were treated with DOX (8 mg/kg) once per week for 4 weeks, with ultrasound imaging every week to assess parameters of ( A ) left ventricular structure and ( B ) cardiac function