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Simultaneously, copper overload can lead to loss of mitochondrial membrane potential (m), which triggers cytochrome c release, further activates the intrinsic apoptosis pathway, accelerates cell death and causes tissue damage (140,141)

"Edetate disodiumbased chelation for patients with a previous myocardial infarction and diabetes: TACT2 randomized clinical trial"

Glycerolipid headgroups control rate and mechanism of superoxide dismutase-1 aggregation and accelerate fibrillization of slowly aggregating amyotrophic lateral sclerosis mutants

PD-1 deficiency increases the comparative levels of certain intermediates and terminal metabolites, such as Glu, N-methylglutamate, N-acetylglutamate (NAG) and ornithine, that participate in Gln metabolism (97)

insH10 (10 times lower expression) and insH21 (9 times lower expression), both of which are believed to be the transposase for the insertion sequence element IS5