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ECM deposition in the Disse space results in the establishment of endodermis and the loss of fenestration in hepatic sinusoidal endothelial cells (HSECs)

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Available data from the APR show no statistically significant difference in the overall risk of major birth defects for EVG, COBI, FTC or TAF compared with the background rate for major birth defects of 2.7% in a U.S

It leads to fewer parietal cells to produce IF and therefore less B12 absorption

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