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In this sense, it has been reported that binding of mutp53 to particular motifs on non-B DNA conformations, confers stability to mutp53, but also makes it more selective for modifying the activity of both transcription factors and chromatin remodelers (Ghler et al., 2005

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Harrington JW, Rosen L, Garnecho A, Patrick PA

nonetheless, certain DNA lesions can escape repair or exceed the repair capacity and eventually accumulate in the genome

In methionine/choline-deficient diet-induced metabolic dysfunction-associated steatohepatitis (MASH), inflammatory monocyte-derived DCs (moDCs) characterized by dual Ly6C + CD11b + CD11c + MHC-II + expression and continuous TNF- production undergo 4-fold expansion during late disease