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Oxidative stress is caused by an imbalance in the rate of free radical formation and detoxification, which ultimately triggers ferroptosis via lipid peroxidation[20]

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It could also protect liver from the altered levels of DNA, RNA, glycogen and cholesterol by 70100%, and hepatic GSH up to 2537%, respectively (441)

Moreover, while some studies indicate beneficial effects of lactylation under certain conditions [20], others suggest potential adverse effects [28], emphasizing the importance of comprehensive investigation to understand these contrasting phenomena

The metabolic profile is very similar to that in column 3 except that now the intracellular SAH concentration does not decline but instead increases by 24% in line with the clinical data